Thromb Haemost 1988; 59(01): 123
DOI: 10.1055/s-0038-1642579
Review Article
Schattauer GmbH Stuttgart

Fibrinolysis after Heart Transplantation

J A Páramo
Haematology Services, University Clinic. University of Navarra, Pamplona, Spain
,
R Arcas
*   Cardiovascular Services, University Clinic. University of Navarra, Pamplona, Spain
,
J Herreros
*   Cardiovascular Services, University Clinic. University of Navarra, Pamplona, Spain
,
B Cuesta
Haematology Services, University Clinic. University of Navarra, Pamplona, Spain
,
R Llorens
*   Cardiovascular Services, University Clinic. University of Navarra, Pamplona, Spain
,
E Rocha
Haematology Services, University Clinic. University of Navarra, Pamplona, Spain
› Author Affiliations
Further Information

Publication History

Received 14 September 1987

Accepted after revision 30 October 1987

Publication Date:
18 April 2018 (online)

 
  • References

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  • 2 Stibbe J, Kluft C, Brommer E JP et al. Enhanced fibrinolytic activity during cardiopulmonary bypass in open-heart surgery in man is caused by extrinsic (tissue-type) plasminogen activator. Eur J Clin Invest 1984; 14: 375-82
  • 3 Mammen EF, Koets MH, Washington BC et al. Hemostasis changes during cardiopulmonary bypass surgery. Sem Thromb Hemostas 1985; 11: 281-92
  • 4 D’Angelo A, Kluft C, Verheijen JH, Rijken DC, Mozzi E, Mannucci PM. Fibrinolytic shut-down after surgery: impairment of the balance between tissue-type plasminogen activator and its specific inhibitor. Eur J Clin Invest 1985; 15: 308-12
  • 5 Páramo JA, Campbell W, Cuesta B, Gómez C, Aranda A, Rocha E. Fibrinolytic response in malignancy. Fibrinolysis 1987; 1: 195-9