Thromb Haemost 2001; 85(03): 562-563
DOI: 10.1055/s-0037-1615625
Letters to the Editor
Schattauer GmbH

Idiopathic Deep Vein Thrombosis in Siblings with Homozygous Factor V Leiden Mutation

Herwig Köppel
1   Department of Medicine, Division of Angiology, Karl Franzens-University Graz, Austria
,
Wilfried Renner
1   Department of Medicine, Division of Angiology, Karl Franzens-University Graz, Austria
,
Gerald Seinost
1   Department of Medicine, Division of Angiology, Karl Franzens-University Graz, Austria
,
Ernst Pilger
1   Department of Medicine, Division of Angiology, Karl Franzens-University Graz, Austria
› Author Affiliations
Further Information

Publication History

Received 18 July 2000

Accepted 14 December 2000

Publication Date:
08 December 2017 (online)

 

 
  • References

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  • 2 Svensson PJ, Dahlbäck B. Resistance to activated protein C as a basis for venous thrombosis. N Engl J Med 1994; 330: 517-22.
  • 3 Koster T, Rosendaal FR, de Ronde H, Briët E, Vandenbroucke JP, Bertina RM. Venous thrombosis due to poor anticoagulant response to activated protein C: Leiden Thrombophilia Study. Lancet 1993; 342: 1503-6.
  • 4 Greengard JS, Eichinger S, Griffin JH, Bauer KA. Variability of Thrombosis among Homozygous Siblings with Resistance to Activated Protein C due to an Arg-Gln Mutation in the Gene for Factor V. N Engl J Med 1994; 331: 1559-62.
  • 5 Eichinger S, Pabinger I, Stümpflen A, Hirschl M, Bialonczyk Ch, Schneider B, Mannhalter Ch, Minar E, Lechner K, Kyrle PA. The Risk of Recurrent Venous Thromboembolism in Patients with and without Factor V Leiden. Thromb Haemost 1997; 77 (Suppl. 04) 624-8.