Impaired Platelet Procoagulant Mechanisms in Patients with Bleeding Disorders

The publisher regrets an error in Table [1] in the above article in Seminars in Thrombosis and Hemostasis, Volume 35, Number 2, 2009, p. 235.

Table [1] with the correct wording in column three (PCA-Related Defect(s)) appears below.

Table 1 Impaired Platelet PCA in Bleeding Disorders Disorder Primary Platelet Defect(s) PCA-Related Defect(s) Other Platelet Defects Screening PCA Abnormality Scott syndrome PS exposure, vesiculation Factor Va, Xa, IXa binding, prothrombinase, tenase (See text) SPT, PF3a Platelet vesiculation defect Vesiculation Vesiculation None SPT Quebec platelet disorder uPA overexpression Prothrombinase, platelet factor V Proteolyzed α-granule proteins Platelet factor V-New York Platelet factor V Prothrombinase None PF3a δ-Storage pool deficiency Dense granules Prothrombinase, ATP, ADP secretion, Ca2+ entry Aggregation PF3a Thrombasthenia GPIIb-IIIa Prothrombinase (±), thrombin potential Aggregation PF3a Bernard-Soulier syndrome GPIb-IX-V Factor XI binding, thrombin binding, fibrin binding Adhesion SPT